Neuromodulation : journal of the International Neuromodulation Society
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Spinal cord stimulation (SCS) is increasingly gaining widespread use as a treatment for chronic pain. A widely used electronic registry could play a pivotal role in improving this complex and cost-intensive treatment. We aimed to construct a comprehensive, universally available data base for SCS. ⋯ We believe that the data base described in this article offers a powerful and versatile data collection tool suited for both clinicians and researchers in the field. The basic data base structure is immediately available on a no-cost basis, and we invite our colleagues to make use of the data base as part of the efforts to further the field of neuromodulation.
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Letter Case Reports
Spinal-peripheral neurostimulation (SPN) for bilateral postherniorrhaphy pain: a case report.
Postherniorrhaphy pain is a not uncommon, and difficult to treat, pain condition. Success with different methods of neurostimulation has been reported in the literature in the last decade. We assess the use of a new modality of neuromodulation--spinal-peripheral neurostimulation (SPN). ⋯ SPN can be an effective treatment for postherniorrhaphy pain resistant to conservative management.
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Occipital nerve stimulation (ONS) may provide relief for refractory headache disorders. However, scant data exist regarding long-term ONS outcomes. ⋯ Randomized controlled long-term studies in specific, intractable, primary headache disorders are indicated.
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Letter Case Reports
Treatment of contact dermatitis associated with spinal cord stimulator pulse generator--technical note.
Expanded polytetrafluoroethylene sheets have been successfully used as a biologic barrier in the treatment of pacemaker contact dermatitis. ⋯ This technique is an effective way of treating contact dermatitis related to implantable pulse generators, without the need for explantation of the neuromodulation system.
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Spinal cord stimulation (SCS) is an efficacious therapy for chronic neuropathic pain whose precise mechanism of action is unclear. Mediators produced by glial and immune cells are now believed to modulate neuronal transmission and promote chronic neuropathic pain. We postulated a relationship between cerebrospinal fluid (CSF) concentrations of neuroimmune mediators and SCS. ⋯ Patients with FBSS have altered CSF levels of BDNF and MCP-1. CSF VEGF correlates with pain and is reduced by SCS. This may offer novel insights into both the mechanism of action of SCS in FBSS and the variation in clinical response that may be encountered.