Articles: vertigo.
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Biography Historical Article
Theophrastus, De Vertigine, Ch. 9, and Heraclitus Fr. 125.
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Randomized Controlled Trial Comparative Study Clinical Trial
Alleviation of induced vertigo. Therapy with transdermal scopolamine and oral meclizine.
Twelve healthy subjects received seven-day treatments on a randomized, double-blind, crossover basis, of a transdermal scopolamine system, oral meclizine, and placebo, separated by one-week intervals. Just prior to each treatment, and on days 1 and 7 of each treatment, subjects received two warm (44 degrees C) caloric irrigations of each external auditory canal. Following each irrigation, subjects rated their vertigo symptoms. ⋯ Vertigo symptoms on day 1 of treatment were significantly less with transdermal scopolamine than oral meclizine or placebo and on day 7 were significantly less with both scopolamine and meclizine than the placebo. On day 1, meclizine did not reduce vertigo symptoms significantly when compared with the placebo. Drowsiness was greater with use of oral meclizine than transdermal scopolamine.
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Nippon Seikeigeka Gakkai Zasshi · Mar 1985
Case Reports[Another new etiology of vertebral artery insufficiency--a case of dynamic entrapment of the vertebral artery by the cervical nerve].
In the etiology of vertebral artery insufficiency, cervical spondylosis with uncal osteophyte, Power's syndrome, A-V malformation and atheromatous narrowing have been recognized. We recently experienced a patient with episodic vertigo and hyposthenia of the left side of the body on rotational movement of the neck. ⋯ With the release and resection of the injured nerve, the patient became free of any vertigo or hyposthenia. Although this kind of etiology involving entrapment-constriction of the vertebral artery by the cervical nerve after trauma has not been reported at all in the literature, we should always keep this in mind when dealing with patients with cervical vertigo after trauma.
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ORL J. Otorhinolaryngol. Relat. Spec. · Jan 1984
Benign paroxysmal vertigo in childhood: a migraine equivalent.
The two main problems in defining and classifying the syndrome of benign paroxysmal vertigo (BPV) in childhood are the vestibular function pattern and the relationship between BPV and migraine. 13 children suffering from this syndrome were submitted to complete otoneurological examination, including caloric and rotational labyrinthine stimulation with ENG recording, and to headache provocation tests with nitroglycerin, histamine and fenfluramine. Vestibular responses were normal in all except 2 cases which presented signs of central vestibular impairment at the level of the vestibulocerebellar pathways. Headache provocation tests were positive in 9 out of 10 children, and in 4 cases they induced a typical vertiginous attack instead of headache. ⋯ During the follow-up period, some children responded positively to migraine treatment. BPV, like paroxysmal torticollis in infancy and the signs of the periodic syndrome, can be considered a migraine equivalent or a migraine precursor and could be due to the same vascular and/or biochemical mechanisms responsible for the migraine. In children, for anatomical or developmental reasons, these mechanisms could selectively affect parts of the brain stem, including the vestibular nuclei and vestibulocerebellar pathways.