Articles: vertigo.
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Acta Otolaryngol Suppl · Jan 1984
Benign paroxysmal vertigo in childhood. Diagnostic significance of vestibular examination and headache provocation tests.
Sixteen children with benign paroxysmal vertigo (BPV) are presented. The great majority had a family history of migraine, neurological and autonomic signs associated with vertiginous attacks, and headache or other sign of the periodic syndrome (motion sickness, cyclic vomiting, abdominal pain) unrelated to the attacks. ⋯ Headache provocation tests with nitroglycerin, histamine and fenfluramine were positive in 9 of the 13 patients examined, and in 4 cases induced a typical vertiginous attack instead of headache. BPV can be considered a migraine precursor or a migraine equivalent, attributable to the same vascular and/or biochemical disturbances responsible for migraine.
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Acta oto-laryngologica · May 1980
The mechanism of physiological height vertigo. I. Theoretical approach and psychophysics.
A theory is presented supporting a geometrical explanation of physiological height vertigo as a 'distance vertigo' created by visual destabilization of posture when the distance between the observer and visible stationary contrasts becomes critically large. Though height vertigo is generally regarded as a psychopathological process, we hypothesize that it might instead result from an intersensory mismatch when visual information is at variance with vestibular and proprioceptive inputs. ⋯ Physiological 'distance vertigo' must be distinguished from psychological 'acrophobia'. Its postural consequences may be ameliorated by strategies gleaned from knowledge of its mechanism such as providing nearby stationary contrasts in the peripheral visual field.
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Clin Otolaryngol Allied Sci · Feb 1980
Case Reports'Costen's syndrome'--correlation or coincidence: a review of 45 patients with temporomandibular joint dysfunction, otalgia and other aural symptoms.
Forty-five patients with temporomandibular joint dysfunction, and otalgia together with other aural symptoms (deafness, tinnitus, pressure/blockage and vertigo) were evaluated clinically and audiometrically. The theoretical mechanisms by which aural symptoms may be produced as a result of temporomandibular joint dysfunction are outlined and discussed in the light of the patients under review. The wide diversity in the incidence of additional aural symptoms apart from otalgia reported in the literature is noted, together with the general lack of full objective audiometric assessment. ⋯ Thus in this series at least 9% of the patients were considered to have other aural symptoms coincidental to temporomandibular joint dysfunction. This is compatible with the relatively common occurrence of both temporomandibular joint dysfunction and aural symptoms in the general population. This study leads us to believe that there is no direct aetiological basis to link temporomandibular joint dysfunction and other aural symptoms apart from otalgia.