• J Cardiothorac Surg · Mar 2014

    Central extracorporeal life support with left ventricular decompression for the treatment of refractory cardiogenic shock and lung failure.

    • Alexander Weymann, Bastian Schmack, Anton Sabashnikov, Christopher T Bowles, Philipp Raake, Rawa Arif, Markus Verch, Ursula Tochtermann, Jens Roggenbach, Aron Frederik Popov, Andre Ruediger Simon, Matthias Karck, and Arjang Ruhparwar.
    • Department of Cardiothoracic Transplantation & Mechanical Circulatory Support, Royal Brompton and Harefield NHS Foundation Trust, Hill End Road, Harefield, Middlesex, UB9 6JH London, UK. weymann.alexander@googlemail.com.
    • J Cardiothorac Surg. 2014 Mar 29; 9: 60.

    BackgroundThe purpose of this prospective study was to evaluate the effects and functional outcome of central extracorporeal life support (ECLS) with left ventricular decompression for the treatment of refractory cardiogenic shock and lung failure.MethodsBetween August 2010 and August 2013, 12 consecutive patients (2 female) with a mean age of 31.6 ± 15.1 years received central ECLS with left ventricular decompression for the treatment of refractory cardiogenic shock and lung failure. Underlying disease was acute cardiac decompensation due to dilated cardiomyopathy (n = 3, 25%), coronary artery disease with acute myocardial infarction (AMI) (n = 3, 25%), and acute myocarditis (n = 6, 50%). We routinely implemented ECLS by cannulating the ascending aorta, right atrium and inserting a left ventricular decompression cannula vent via the right superior pulmonary vein.ResultsAll patients were successfully bridged to either recovery (n = 3, 25%), long-term biventricular support (n = 6, 50%) or cardiac transplantation (n = 3, 25%). Seven patients (58.3%) were discharged after a mean hospital stay of 42 ± 11.9 days. The overall survival from ECLS implantation to the end of the study was 58.3%. The cumulative ICU stay was 23.1 ± 9.6 days. The length of support was 8.0 ± 4.3 days (range 3-17 days).ConclusionsWe strongly recommend left ventricular decompression in refractory cardiogenic shock and lung failure to avoid pulmonary edema, left heart distension and facilitate myocardial recovery.

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