• J Orofac Pain · Jan 2012

    Occlusal factors are not related to self-reported bruxism.

    • Daniele Manfredini, Corine M Visscher, Luca Guarda-Nardini, and Frank Lobbezoo.
    • Department of Maxillofacial Surgery, University of Padova, Padova, Italy. daniele.manfredini@tin.it
    • J Orofac Pain. 2012 Jan 1;26(3):163-7.

    AimsTo estimate the contribution of various occlusal features of the natural dentition that may identify self-reported bruxers compared to nonbruxers.MethodsTwo age- and sex-matched groups of self-reported bruxers (n = 67) and self-reported nonbruxers (n = 75) took part in the study. For each patient, the following occlusal features were clinically assessed: retruded contact position (RCP) to intercuspal contact position (ICP) slide length (< 2 mm was considered normal), vertical overlap (< 0 mm was considered an anterior open bite; > 4 mm, a deep bite), horizontal overlap (> 4 mm was considered a large horizontal overlap), incisor dental midline discrepancy (< 2 mm was considered normal), and the presence of a unilateral posterior crossbite, mediotrusive interferences, and laterotrusive interferences. A multiple logistic regression model was used to identify the significant associations between the assessed occlusal features (independent variables) and self-reported bruxism (dependent variable).ResultsAccuracy values to predict self-reported bruxism were unacceptable for all occlusal variables. The only variable remaining in the final regression model was laterotrusive interferences (P = .030). The percentage of explained variance for bruxism by the final multiple regression model was 4.6%. This model including only one occlusal factor showed low positive (58.1%) and negative predictive values (59.7%), thus showing a poor accuracy to predict the presence of self-reported bruxism (59.2%).ConclusionThis investigation suggested that the contribution of occlusion to the differentiation between bruxers and nonbruxers is negligible. This finding supports theories that advocate a much diminished role for peripheral anatomical-structural factors in the pathogenesis of bruxism.

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