• Clinical calcium · Mar 2005

    [Experimental studies of lumbar spinal stenosis].

    • Miho Sekiguchi and Shin-ichi Kikuchi.
    • Fukushima Medical University School of Medicine, Department of Orthopaedic Surgery.
    • Clin Calcium. 2005 Mar 1; 15 (3): 51-6.

    AbstractNeurophysiologic function and ischemia causes neurogenic symptoms in lumbar spinal stenosis. Neurophysiologic function and potential therapeutic agents were investigated using a chronic cauda equina compression model that was developed according to a clinical pathogenesis. In the chronic cauda equina compression model, nerve conduction velocity and blood flow in spinal nerves decreased but mechanical allodynia was not induced. Prostaglandins and 5-HT receptor antagonist effected to improve blood flow in nerve roots after cauda equina compression;therefore, they might be potential agents for lumbar spinal stenosis.

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