• Front Mol Neurosci · Jan 2018

    Review

    Autoimmune Neurological Conditions Associated With Zika Virus Infection.

    • Yeny Acosta-Ampudia, Diana M Monsalve, Luis F Castillo-Medina, Yhojan Rodríguez, Yovana Pacheco, Susan Halstead, Hugh J Willison, Juan-Manuel Anaya, and Carolina Ramírez-Santana.
    • Center for Autoimmune Diseases Research, School of Medicine and Health Sciences, Universidad del Rosario, Bogota, Colombia.
    • Front Mol Neurosci. 2018 Jan 1; 11: 116.

    AbstractZika virus (ZIKV) is an emerging flavivirus rapidly spreading throughout the tropical Americas. Aedes mosquitoes is the principal way of transmission of the virus to humans. ZIKV can be spread by transplacental, perinatal, and body fluids. ZIKV infection is often asymptomatic and those with symptoms present minor illness after 3 to 12 days of incubation, characterized by a mild and self-limiting disease with low-grade fever, conjunctivitis, widespread pruritic maculopapular rash, arthralgia and myalgia. ZIKV has been linked to a number of central and peripheral nervous system injuries such as Guillain-Barré syndrome (GBS), transverse myelitis (TM), meningoencephalitis, ophthalmological manifestations, and other neurological complications. Nevertheless, mechanisms of host-pathogen neuro-immune interactions remain incompletely elucidated. This review provides a critical discussion about the possible mechanisms underlying the development of autoimmune neurological conditions associated with Zika virus infection.

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