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Critical Care Posters IIISESSION TYPE: Poster PresentationsPRESENTED ON: Saturday, March 22, 2014 at 01:15 PM - 02:15 PMPURPOSE: We previously demonstrated in a clinically relevant murine model of sepsis, that animals with left ventricular (LV) dilation had better cardiovascular performance and increased survival. Using the same model of sepsis, we sought to determine if survivors have an early phenotype different from non-survivors with different resuscitation regiment. ⋯ The following authors have nothing to disclose: François Dépret, Fabien Picard, Sergio Zanotti-Cavazzoni, Steven HollenbergNo Product/Research Disclosure Information.
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Critical Care Posters IIISESSION TYPE: Poster PresentationsPRESENTED ON: Saturday, March 22, 2014 at 01:15 PM - 02:15 PMPURPOSE: To identify risk factor on admission in patients with acute GIH, to predict need of critical care monitoring. ⋯ The following authors have nothing to disclose: Amitesh Agarwal, Marlow Hernandez, Gustavo FerrerNo Product/Research Disclosure Information.
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Surgery Case Report Posters ISESSION TYPE: Case Report PosterPRESENTED ON: Sunday, March 23, 2014 at 01:15 PM - 02:15 PMINTRODUCTION: Incidence of hiatal hernias (HH) increases with age. Approximately 60% of individuals aged 50 or older have a HH. [1] A giant HH is a hernia that includes at least 30% of the stomach in the chest, although a uniform definition does not exist; most commonly, a giant HH is a type III hernia with a sliding and paraesophageal component [2]. Patients with a giant HH generally present with pain, heartburn or a history of heartburn, dysphagia, vomiting, and anemia. We present a case of giant HH presenting with hematemesis. ⋯ This case illustrates the need to consider giant HH in the presence of hematemesis. Early recognition of this rare entity can lead to early surgical intervention.Reference #1: Goyal Raj K, "Chapter 286. Diseases of the Esophagus". Harrison's Principles of Internal Medicine, 17e.Reference #2: Mitiek, MO "Giant Hiatal Hernia" Annl Thor Surg 2010 Jun;89(6):S2168-73.Reference #3: Landreneau RJ et al. Management of paraesophageal hernias Surg Clin North Am 2005;85:411-432DISCLOSURE: The following authors have nothing to disclose: Anita Rajagopal, Anthony AsciotiNo Product/Research Disclosure Information.
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ILD Case Report Posters IISESSION TYPE: Case Report PosterPRESENTED ON: Sunday, March 23, 2014 at 01:15 PM - 02:15 PMINTRODUCTION: The Antisynthetase syndrome is a rare entity, its comprises of a triad of inflammatory myopathy, diffuse interstitial lung disease, and small joint involvement. Serum auto antibodies to aminoacyl transfer RNA synthetases (anti-ARS) especially anti Jo-1 antibody present in most of the cases. Interstitial lung disease is an important diagnostic criteria and a major determinant of mortality of this syndrome. We are reporting a case of ASSCASE PRESENTATION: 48 year old female patient presented in OPD with complained of persistent dry cough and grade III dyspnea on exertion, generalised muscular pain and weakness. On examination she had mild anaemia, Pulse rate 96/mt BP 140/90 no lymphadenopathy, no clubbing. Respiratory system examination revealed crepitation in infrascapular area on both the side.Her hand were rough with creaky skin, crease of palm were dark suggestive of classical mechanics hand (figure 1). The movements of both the thumb were restricted .She was giving the history of joint pain and of Reynaud phenomenon.Investigations revealed,Hb 9.5 % gm, serum ANA was positive, Anti Ds DNA was negative, and C3 C4 and Rheumatoid factor was also negative. Transbronchial lung biopsy revealed nonspecific fibrosis and no evidence of granulomatus disorders. HRCT scan thorax was suggestive of bilateral lower lobe interstitial fibrosis in form of septal thickening, traction bronchieactasis and early honeycombing (figure 3&4). Spirometry revealed restrictive pattern . In the serum anti JO-1 was strongly positive. X-ray of the hand revealed subluxations of metacarpophalengeal joints in both the hand and progressive ankylosis of the same joint (figure2). Clinic radiological findings and serological findings were suggestive of interstitial lung disease due to AAS. ⋯ The Antisynthetase syndrome is a rare clinical entity, having multisystem involvement .It is comprises of a triad of inflammatory myopathy, diffuse interstitial lung disease, and small joint involvement. If this clinical entity diagnosed on time and treated adequately , then irreversible changes can be prevented.Reference #1: Love LA, Leff RH, Fraser DD , Targoff IN, Dalakas M, Plotz PH. Miller FW.A new approach to the classification of idiopathic inflammatory myopathy:myositis-specific autoantibody define useful homogeneous patient groups.Medicine(Baltimore) 70:360-74;1991Reference #2: Marguerie C, Bunn CC, Beynon HL, Bernstein RM, Hughes JM, So AK et al. Polymyositis, pulmonary fibrosis and autoantibodies to aminoacyl-Trna synthetase enzymes. Q J Med 1990; 77(282):1019-38.Reference #3: Chinoy H, Salway F, Fertig N, Shephard N, Tait BD, Thomson W et al. In adult onset myositis, the presence of interstitial lung disease and myositis specific/associated antibodies are governed by HLA class II haplotype, rather than by myositis subtype. Arthritis Res Ther 2006; 8(1):R13DISCLOSURE: The following authors have nothing to disclose: Ashok Singh, Raghvendra JaiswalNo Product/Research Disclosure Information.
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Pleural Case Report PostersSESSION TYPE: Case Report PosterPRESENTED ON: Sunday, March 23, 2014 at 01:15 PM - 02:15 PMINTRODUCTION: Spontaneous pneumomediastinum (SPM) is an unusual condition. It is reported to present in one in 1,000 to one in 40,000 ED referrals. It is characterized by the presence of interstitial air in the mediastinum without any apparent precipitating factor. It is seen after intrathoracic pressure changes leads to alveolar rupture and dissection of air along the tracheobronchial tree. We report a case of a young male who presented with a spontaneous pneumomediastinum. ⋯ The following authors have nothing to disclose: Vanessa Yap, Debapriya DattaNo Product/Research Disclosure Information.